Independent Women? Social class and Social Policy after the Gender Revolution in the West

The gender revolution of the mid-20th century resulted in extensive changes in social, economic, and policy realms. With their unprecedented entrance into the paid labor market, women moved toward greater economic independence. However, it remains unclear whether these changes in women’s economic well-being resulted in positive change for women across the class spectrum. The large-scale entrance of women into paid labor was not accompanied by men’s equal take up in unpaid domestic work. Thus, women found themselves struggling to balance work and family life and as a result, gender inequality in employment outcomes persists. In this dissertation I explore the claim that in the absence of government provided support to reconcile work and family, less privileged women (i.e. women of lower social class) face greater challenges in balancing work and family life than more privileged women. If women’s responsibility for unpaid domestic work is causing women to trail behind men in the paid labor market, then women’s possibility to outsource unpaid domestic labor will affect their participation in the paid labor market. There is a greater ability among women of higher social class to outsource domestic labor or use market substitutes. Therefore, in countries that have not implemented generous work-family reconciliation policies I expect less privileged women to be more economically dependent on their male partners than more privileged women. To assess these expectations, I revisit traditional analysis of gender inequality and develop a measure of economic dependence to assess women’s economic well-being across the class spectrum in 22 Western countries. While most evaluations of women’s agency have assumed that workforce participation is itself a measure of independence, this dissertation goes beyond this assumption and assesses women’s economic dependence by measuring women’s share of the couple’s joint income. I use a comprehensive earnings measure, augmented earnings, which consists of labor market earnings, as well as public and private transfers, and capital income. The analysis is framed in terms of national level work-family reconciliation policies, thus revealing how different policies influence not only women’s economic independence, but also the distribution of that independence across social classes. I first provide an extensive cross-national descriptive analysis of women’s employment outcomes (e.g. employment rates, work intensity, and female to male earnings ratios), and women’s share of the couple’s joint income. Both employment outcomes and economic dependence are analyzed by family type and class status (class is measured with household income and education). The findings from these descriptive chapters reveal three major gender gaps in modern Western society: 1) in employment rates, 2) hours among the employed, 3) and earnings per hour. I find that even when accounting for transfers, in all countries, women contribute on average less than 50% to their household income and in some instances as low as 20%. However, the degree of dependency does vary by class and country. Overall countries with more generous work-family reconciliation policies exhibit more class equality in women’s economic independence. Following the descriptive analysis, I employ a random effect within-between model (REWB) to explore whether the effect of class (measured with education) on women’s economic dependence is moderated by work-family reconciliation policy. Specifically, I analyze the cross-level interaction effect of class and work-family reconciliation policy on women’s augmented earnings relative to the median man in their country. I run the regression models for all women, not just those in employment. Thus, my dependent variable, relative earnings, is a composite index meant to capture the three major gender gaps: in employment, earnings, and work intensity. Findings reveal that being less educated significantly decreases women’s relative earnings, but the association between education and relative earnings is moderated by work-family reconciliation policy. In countries with an “ideal” maternity leave, exclusive paternity leave, more childcare support and more extensive working time regulations, the negative effect of low education on relative earnings is lessened. First, in countries that have these policies in place, women of all levels of education do better than in countries with less generous work-family reconciliation policies. Second, there is also less class inequality among women in countries with these policies. That is, the gap in relative earnings between women of different levels of education is smaller than in countries without these policies. Finally, I find that the moderating effect of work-family reconciliation policies can operate through more than one pathway, i.e., via employment rates, hours worked, or earnings per hour

Imprint of Climate Change on Pan-Arctic Marine Vegetation

The Arctic climate is changing rapidly. The warming and resultant longer open water periods suggest a potential for expansion of marine vegetation along the vast Arctic coastline. We compiled and reviewed the scattered time series on Arctic marine vegetation and explored trends for macroalgae and eelgrass (Zostera marina). We identified a total of 38 sites, distributed between Arctic coastal regions in Alaska, Canada, Greenland, Iceland, Norway/Svalbard, and Russia, having time series extending into the 21st Century. The majority of these exhibited increase in abundance, productivity or species richness, and/or expansion of geographical distribution limits, several time series showed no significant trend. Only four time series displayed a negative trend, largely due to urchin grazing or increased turbidity. Overall, the observations support with medium confidence (i.e., 5–8 in 10 chance of being correct, adopting the IPCC confidence scale) the prediction that macrophytes are expanding in the Arctic. Species distribution modeling was challenged by limited observations and lack of information on substrate, but suggested a current (2000–2017) potential pan-Arctic macroalgal distribution area of 820.000 km2 (145.000 km2 intertidal, 675.000 km2 subtidal), representing an increase of about 30% for subtidal- and 6% for intertidal macroalgae since 1940–1950, and associated polar migration rates averaging 18–23 km decade–1. Adjusting the potential macroalgal distribution area by the fraction of shores represented by cliffs halves the estimate (412,634 km2). Warming and reduced sea ice cover along the Arctic coastlines are expected to stimulate further expansion of marine vegetation from boreal latitudes. The changes likely affect the functioning of coastal Arctic ecosystems because of the vegetation’s roles as habitat, and for carbon and nutrient cycling and storage. We encourage a pan-Arctic science- and management agenda to incorporate marine vegetation into a coherent understanding of Arctic changes by quantifying distribution and status beyond the scattered studies now available to develop sustainable management strategies for these important ecosystems.publishedVersio

Confronting the unknown—Nursing surveillance of COVID-19-infected patients through remote telephone calls and in an on-site urgent clinic

Funding Information: University of Iceland Research Fund. We wish to thank Runólfur Pálsson, MD, Professor and Director of Internal Medicine and Rehabilitation Services, Sigríður Gunnarsdóttir, Chief Nursing Officer for support in conducting this study, Drífa Katrín Guðmundsdóttir Blöndal and Stefanía Bergsdóttir for transcribing the interviews and giving an insightful view on the data analysis and Ásvaldur Kristjánsson for assistance with conducting the focus group interviews. Publisher Copyright: © 2022 The Authors. Journal of Advanced Nursing published by John Wiley & Sons Ltd.Aim: To describe nursing surveillance of coronavirus disease 2019 (COVID-19)-infected patients through remote telephone calls and in an on-site urgent clinic during the first wave of the pandemic as experienced by nurses providing the care. Design: Qualitative descriptive study. Methods: Data were collected through seven semi-structured, audio-recorded, focus group interviews with 24 nurses. Interviews were conducted in May and June 2020, transcribed and analysed using deductive and inductive content analysis into an overarching category, main categories and subcategories. Reporting followed the COREQ guidelines. Results: Nurses relied on intensive listening when assessing and caring for COVID-19-infected patients. They realized that the patients had complex needs for nursing and healthcare which was beyond the scope of a tentatively prescribed assessment scheme. They designed their care to ensure holistic care, reflected in the overarching category, ‘Confronting an unfamiliar health condition in unprecedented circumstances’ and the categories: ‘Digging into the unknown’ and ‘Ensuring holistic nursing care’. The category ‘Contributing to averting catastrophe’ reflects the wealth of knowledge, support and experience that the nurses used to independently deliver care, albeit in interdisciplinary collaboration, working to their greatest potential. They were proud of the significance of their work. Conclusion: Novel nursing surveillance through remote telephone calls and in an on-site urgent care clinic delivered to COVID-19 patients self-managing at home resulted in holistic nursing care during the first wave of the pandemic. This has relevance for professionalism in nursing. Impact: Findings give a unique insight into nursing surveillance of COVID-19-infected patients provided through telephone calls and in on-site urgent care clinics. The potential of intensive listening as conducted in the study suggests that it may be feasible to assess and holistically take care of COVID-19-infected patients, and other patient groups as well, with this form of healthcare. This has relevance for healthcare beyond crisis management during pandemics. Patient or Public Contribution: There was no patient or public contribution as the study only concerned the providers of the service, i.e. the nurses themselves.Peer reviewe

Genome-wide mapping of cystitis due to Streptococcus agalactiae and Escherichia coli in mice identifies a unique bladder transcriptome that signifies pathogen-specific antimicrobial defense against urinary tract infection

The most common causes of urinary tract infections (UTIs) are Gram-negative pathogens such as Escherichia coli; however, Gram-positive organisms, including Streptococcus agalactiae, or group B streptococcus (GBS), also cause UTI. In GBS infection, UTI progresses to cystitis once the bacteria colonize the bladder, but the host responses triggered in the bladder immediately following infection are largely unknown. Here, we used genome-wide expression profiling to map the bladder transcriptome of GBS UTI in mice infected transurethrally with uropathogenic GBS that was cultured from a 35-year-old women with cystitis. RNA from bladders was applied to Affymetrix Gene-1.0ST microarrays; quantitative reverse transcriptase PCR (qRT-PCR) was used to analyze selected gene responses identified in array data sets. A surprisingly small significant-gene list of 172 genes was identified at 24 h; this compared to 2,507 genes identified in a side-by-side comparison with uropathogenic E. coli (UPEC). No genes exhibited significantly altered expression at 2 h in GBS-infected mice according to arrays despite high bladder bacterial loads at this early time point. The absence of a marked early host response to GBS juxtaposed with broad-based bladder responses activated by UPEC at 2 h. Bioinformatics analyses, including integrative system-level network mapping, revealed multiple activated biological pathways in the GBS bladder transcriptome that regulate leukocyte activation, inflammation, apoptosis, and cytokine-chemokine biosynthesis. These findings define a novel, minimalistic type of bladder host response triggered by GBS UTI, which comprises collective antimicrobial pathways that differ dramatically from those activated by UPEC. Overall, this study emphasizes the unique nature of bladder immune activation mechanisms triggered by distinct uropathogens

The academic–vocational divide in three Nordic countries : implications for social class and gender

In this study we examine how the academic–vocational divide is manifested today in Finland, Iceland and Sweden in the division between vocationally (VET) and academicallyoriented programmes at the upper-secondary school level. The paper is based on a critical re-analysis of results from previous studies; in it we investigate the implications of this divide for class and gender inequalities. The theoretical lens used for the synthesis is based on Bernstein´s theory of pedagogic codes. In the re-analysis we draw on previous studies of policy, curriculum and educational praxis as well as official statistics. The main conclusions are that contemporary policy and curriculum trends in all three countries are dominated by a neo-liberal discourse stressing principles such as “market relevance” and employability. This trend strengthens the academic–vocational divide, mainly through an organisation of knowledge in VET that separates it from more general and theoretical elements. This trend also seems to affect VET students’ transitions in terms of reduced access to higher education, particularly in male-dominated programmes. We also identify low expectations for VET students, manifested through choice of textbooks and tasks, organisation of teacher teams and the advice of career counsellors.Peer reviewe

Pathogen Specific, IRF3-Dependent Signaling and Innate Resistance to Human Kidney Infection

The mucosal immune system identifies and fights invading pathogens, while allowing non-pathogenic organisms to persist. Mechanisms of pathogen/non-pathogen discrimination are poorly understood, as is the contribution of human genetic variation in disease susceptibility. We describe here a new, IRF3-dependent signaling pathway that is critical for distinguishing pathogens from normal flora at the mucosal barrier. Following uropathogenic E. coli infection, Irf3−/− mice showed a pathogen-specific increase in acute mortality, bacterial burden, abscess formation and renal damage compared to wild type mice. TLR4 signaling was initiated after ceramide release from glycosphingolipid receptors, through TRAM, CREB, Fos and Jun phosphorylation and p38 MAPK-dependent mechanisms, resulting in nuclear translocation of IRF3 and activation of IRF3/IFNβ-dependent antibacterial effector mechanisms. This TLR4/IRF3 pathway of pathogen discrimination was activated by ceramide and by P-fimbriated E. coli, which use ceramide-anchored glycosphingolipid receptors. Relevance of this pathway for human disease was supported by polymorphic IRF3 promoter sequences, differing between children with severe, symptomatic kidney infection and children who were asymptomatic bacterial carriers. IRF3 promoter activity was reduced by the disease-associated genotype, consistent with the pathology in Irf3−/− mice. Host susceptibility to common infections like UTI may thus be strongly influenced by single gene modifications affecting the innate immune response

The Toll-Like Receptor 4 (TLR4) Variant rs2149356 and Risk of Gout in European and Polynesian Sample Sets

Deposition of crystallized monosodium urate (MSU) in joints as a result of hyperuricemia is a central risk factor for gout. However other factors must exist that control the progression from hyperuricaemia to gout. A previous genetic association study has implicated the toll-like receptor 4 (TLR4) which activates the NLRP3 inflammasome via the nuclear factor-κB signaling pathway upon stimulation by MSU crystals. The T-allele of single nucleotide polymorphism rs2149356 in TLR4 is a risk factor associated with gout in a Chinese study. Our aim was to replicate this observation in participants of European and New Zealand Polynesian (Māori and Pacific) ancestry. A total of 2250 clinically-ascertained prevalent gout cases and 13925 controls were used. Non-clinically-ascertained incident gout cases and controls from the Health Professional Follow-up (HPFS) and Nurses Health Studies (NHS) were also used. Genotypes were derived from genome-wide genotype data or directly obtained using Taqman. Logistic regression analysis was done including age, sex, diuretic exposure and ancestry as covariates as appropriate. The T-allele increased the risk of gout in the clinically-ascertained European samples (OR = 1.12, P = 0.012) and decreased the risk of gout in Polynesians (OR = 0.80, P = 0.011). There was no evidence for association in the HPFS or NHS sample sets. In conclusion TLR4 SNP rs2143956 associates with gout risk in prevalent clinically-ascertained gout in Europeans, in a direction consistent with previously published results in Han Chinese. However, with an opposite direction of association in Polynesians and no evidence for association in a non-clinically-ascertained incident gout cohort this variant should be analysed in other international gout genetic data sets to determine if there is genuine evidence for association